MolTransport :


Entities	CAMK1 ---> HDAC5
References	3
Connectivity	2
Effect	unknown
Mechanism	export
Owner	Public
URN	urn:agi-MolTransport:inout-urn:agi-llid:8536:out-urn:agi-llid:10014::unknown:export


Journal Link	http://www.jbc.org/cgi/content/full/276/37/35042
Journal	J. Biol. Chem
Source	Curated
MedLine Reference	11470791:1229
Sentence	Recently, it was reported that an active form of calcium/calmodulin-dependent protein kinase I (CaMKI) is capable of promoting HDAC5???· 14-3-3 binding and leads to HDAC5 export in cultured cells ( 17 ).
Journal Reference	v276 i37 p35042 (2001)


Journal Link	http://www.jbc.org/cgi/content/full/276/5/2975
Journal	J. Biol. Chem
Source	Curated
MedLine Reference	11096122:1143
Sentence	Association of MEF2 with HDAC4/5 can also be inhibited by cotransfection with a constitutively active fragment of CaMKI/IV, which may provide an indirect mechanism for CaMKI/IV enhancement of MEF2 transcription ( 53 ).
CellType	t cell
Journal Reference	v276 i5 p2975 (2001)


Journal Link	http://jcs.biologists.org/cgi/content/full/114/24/4477
Journal	Cell Sci
Organism	mouse
CellLineName	10T1/2
Source	Curated
MedLine Reference	11792813:1030
Sentence	Export of HDAC5 to the cytoplasm, following phosphorylation of specific serine residues by activated CAMKI, was shown to be essential for efficient myogenic conversion of 10T1/2 fibroblasts by exogenous MyoD ( McKinsey et al., 2000 ), an event that requires MEF2 ( Molkentin et al., 1995 ) and histone acetylase (HAT) activity ( Sartorelli et al., 1997 ).
CellType	fibroblast
Journal Reference	v114 i24 p4477 (1997)